ASD Device Closure in Hyderabad
An atrial septal defect (ASD) is one of the most common congenital heart conditions — a hole in the wall between the heart's two upper chambers that some people are born with and others discover only in adulthood. ASD device closure is a minimally invasive catheter-based procedure that permanently seals this defect without any chest incision or open-heart surgery. At AIG Hospitals, Gachibowli, Dr. Bhishma Chowdary performs ASD device closure as part of a dedicated structural heart programme.
At a Glance
| Procedure type | Structural heart intervention — catheter-based defect closure |
| Condition treated | Atrial septal defect (ASD) — hole in the interatrial septum |
| Device used | Amplatzer Septal Occluder or equivalent double-disc device |
| Duration | 1 to 2 hours |
| Anaesthesia | Conscious sedation or general anaesthesia. Guided by TOE. |
| Hospital stay | 1 to 2 nights |
| Recovery | Light activities within 1 week. Full activity at 4 to 6 weeks. |
| Follow-up | Echo at 1 month and 6 months. Antibiotic prophylaxis 6 months. |
| Performed by | Dr. Bhishma Chowdary, DM Cardiology, AIG Hospitals Hyderabad |
| Appointment | +91-9000352998 |
Understanding the atrial septum and ASD
The heart has four chambers — two upper chambers (atria) and two lower chambers (ventricles). The wall separating the left and right atria is called the interatrial septum. In normal fetal development, a small opening called the foramen ovale exists in this wall, allowing blood to bypass the lungs before birth. This opening closes naturally within the first few months of life in most people.
An atrial septal defect occurs when this closure is incomplete, leaving a persistent hole of varying size. Blood then flows from the higher-pressure left atrium across to the right atrium — called a left-to-right shunt. This extra volume overloads the right side of the heart and the pulmonary circulation, forcing the heart and lungs to handle more blood than they should. Over years, this excess workload leads to progressive right heart enlargement, reduced exercise tolerance, arrhythmias, and eventually pulmonary hypertension if untreated.